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Pathway analyses of differential genomic occupancy for both epigenetic marks showed enrichment in several inflammatory pathways, including TNF superfamily cytokine production, IL-6-mediated signaling, LPS-mediated signaling, Toll-like receptor signaling, cytokine production, type II interferon production and chemokine-mediated signaling (Extended Data Fig

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10.1007/s00204-017-2036-4 Arch

1) Pro-angiogenic signaling (VEGFR2 Akt eNOS) In cell and animal models, BPC-157 increases VEGFR2 expression/internalization and activates downstream Akt-eNOS signaling, supporting endothelial tube formation and blood-flow recovery after ischemia

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